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Dysprosody, dysarthria, are exaggerated in PD); (2) overall hypersecretion of GH with secondary inhi- bition of GH secretion to secretory stimuli; (3) traving abnor- mally enhanced negative feedback system, that is, in- creased pituitary sensitivity to the inhibitory effects of GH or somatomedin-C; (4) subsensitivity of the pituitary to the secretory effect of GRF or reduced GRF; (5) failure of cloni- dine to reduce the inhibitory effect of somatostatin neurons on the pituitary; and (6) abnormal function of a host of other factors previously enumerated.

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